MT2A promotes angiogenesis in chronically ischemic brains through a copper–mitochondria regulatory mechanism

Table 1 Subgrouping of HUVECs

Groups	Procedure
Control group	Treated with ES in a normoxic environment
Nor group	Treated with ES and CuCl₂ (1 µM) in a normoxic environment
Hyp group	Only treated with ES in a hypoxic environment
Hyp^CPO group	Treated with ES and CuCl₂ (1 µM) in a hypoxic environment

HUVECs: human umbilical vein endothelial cells; ES: elesclomol; Nor: normoxia; Hyp: hypoxia; CPO: copper overload

ISSN: 1479-5876